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Pinal County Public Health & State Confirm H1N1-related Death
Pinal County Public Health officials have received confirmation that a 64-year-old woman who died last week was positive for the H1N1 virus. The woman had underlying health conditions and was being treated for pneumonia at the time of her death.
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Heart Disease Researcher Wins Scholarship
Accredited Practising Dietitian and PhD student Janice Sangster has been awarded the Dietitians
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Missouri Legislature Passes Bills That Impose Levies On Health Care Providers To Generate Additional Federal Medicaid Funding
Missouri lawmakers have approved two bills (HB 740 and SB 307) that extend or create health care provider taxes or certification fees as part of an effort to draw in more federal Medicaid matching dollars, the AP/Ann Arbor News reports. HB 740 would create a provider tax for ambulances and in-home care providers and a certification fee for certain mental health care providers. SB 307 would extend until 2011 the expiration date on provider taxes on pharmacies, and Medicaid managed care and intermediate care facilities for the mentally disabled. The taxes and fees are collected to generate additional federal Medicaid dollars, which are then redistributed to the providers. Existing taxes levied on industries such as hospitals, nursing homes, prescription drug providers and Medicaid managed care companies generated nearly $1.1 billion last fiscal year, resulting in an additional $1.8 billion in matching federal Medicaid funds, according to data from the Missouri Department of Social Services and Department of Mental Health (AP/Ann Arbor News, 5/12).
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Protecting The Heart With Glucocorticoid Drugs

Glucocorticoids are steroid hormones that have numerous functions; for example, they regulate the response to stress and suppress inflammation. Synthetic glucocorticoids are used clinically in many situations, most famously to treat asthma, allergies, and autoimmunity. They have also been shown in animals and humans to help protect the heart from the damaging effects of heart attack, and this has been attributed to their anti-inflammatory effects. However, Motoaki Sano and colleagues, at Keio University School of Medicine, Japan, have now determined another mechanism by which glucocorticoids protect rodent hearts from the damaging effects of heart attack. Specifically, glucocorticoids, acting via the glucocorticoid receptor (GR), induced mouse and rat heart muscle cells to produce PGD2, and this was responsible for the ability of glucocorticoids to reduce damage to mouse hearts in both an ex vivo and an in vivo model of heart attack. The authors therefore suggest that GR-selective glucocorticoids might be more beneficial to humans following heart attack than glucocorticoids that activate both GR and the MR protein, activation of which occurs in response to stress and might have unwanted consequences. TITLE: Glucocorticoid protects rodent hearts from ischemia/reperfusion injury by activating lipocalin-type prostaglandin D synthase-derived PGD2 biosynthesis AUTHOR CONTACT: Motoaki Sano Keio University School of Medicine, Tokyo, Japan. View the PDF of this article at: https://www.the-jci.org/article.php?id=37413 Karen Honey Journal of Clinical Investigation


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